When the coronavirus SARS-co-v2, a devastating communicable disease, descended upon the global population in December 2019, it became clear early on that there was a wide spectrum of illness, ranging from almost nothing to diarrhea, severe pneumonia, stroke, and death. By May of 2020, it became even more clear that there are particular conditions (“comorbidities”) that markedly increase the disease severity and death rate, and that these conditions had in common an immune system dysfunction with the shared characteristic of chronic inflammation. We have learned that people with chronic inflammation have higher levels of furin and that furin acts as a facilitator to allow better binding of the viral spike protein to our ACE2 receptors. Harboring chronic inflammation turns us into something like Velcro, attracting more virus, secreting more furin, and allowing more severe disease states to evolve.  Read more